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Jump-Start Your Metabolism: 5 Ways to Burn More Calories
Hoping to shed pounds or keep them off? Take 5 steps to rev your engine a little faster

04/08/2019 By Michelle Lee with Craig Weatherby

Hoping to shed a few pounds, or maintain your current weight?

One oft-overlooked strategy is to keep your metabolic engine revving at a relatively high rate.

Your basal metabolic rate or BMR is the number of calories your body typically burns when you’re at rest.

Once you stop growing bone, around age 25, your BMR begins a slow and steady decline of about two percent a year.

So as you age, you need fewer and fewer calories just to maintain your current weight, with the extras getting stored as hard-to-lose, potentially pro-inflammatory body fat.

Cutting calories below your basic needs will just make your BMR plummet.

When you don’t eat enough calories to fuel your BMR, your body will consume muscle — and some fat — to get the missing calories.

And as muscle mass drops, so does your metabolic rate. Low-calorie diets tend not to work in part because they consume calorie-burning muscle … while triggering appetite-stimulating hormones.

The evidence suggests two keys to losing or maintaining weight:

  1. Eat just enough to avoid hunger
  2. Give your metabolism a jump-start

And most experts agree on five simple, powerfully healthful ways to kick your metabolism into higher gear.

1. Don’t overdo low-calorie dieting
Drastic dieting is counterproductive, in part because it drives your BMR downwards.

Depriving your body and brain of their minimal calorie needs reduces your metabolic rate enough to derail attempts to burn body fat.

The key is to consume enough calories to meet your resting metabolic rate, so you get enough fuel (calories) to avoid the body’s survival-driven response: a counterproductive drop in your BMR.

How many calories do you need to maintain your BMR? The National Institutes of Health offers a Body Weight Planner tool that provides an answer based on your personal characteristics and your weight loss or maintenance goals.

Entering your information produces three key numbers:

  1. Daily calories needed to maintain your weight
  2. Daily calories needed to reach your weight goal.
  3. Daily calories needed to maintain your weight goal, which typically falls between the first two numbers.

2. Build and maintain muscle as you age
Simply put, the more muscle you’ve got, the more calories you’ll burn.

At rest, every pound of muscle burns about six calories, while a pound of fat burns only two.

So adding muscle will boost your metabolism even when you’re sitting on the couch or at your desk.

As we said, your metabolism slows as you age, and you lose muscle mass over time. Women lose muscle nearly twice as quickly, due to menopause-related hormonal shifts.

You can build muscle with “resistance” exercises, such as weightlifting, weight machines, or at-home exercises that leverage your own body weight.

Not everyone can or wants to lift weights, buy weight machines, or use gyms for resistance workouts.

Fortunately, “bodyweight” resistance training — i.e., exercises that leverage your own body weight to build muscle — can work really well.

Bodyweight exercises include things like squats, lunges, abdominal crunches, pushups, pullups or step-ups – which require only a floor with a rug or mat.

You’ll find 20 very short videos of bodyweight exercises at Live Strong.com. (See a trainer to learn safe technique, especially if you have bone/joint or other health issues.)

Adding just one or two weekly resistance training sessions can greatly help to preserve and build muscle, reliably raising your basal metabolic rate.

For example, State University of New York researchers examined the effects of resistance training — versus combined resistance training and walking — in moderately obese, previously sedentary women.

After 20 weeks/five months of exercise, the resistance-training group saw significant gains in strength, muscle mass, and in basal metabolic rate — even though its members didn’t lose body fat.

Exercise also helps shed obesity-promoting “white” fatty tissue — and omega-3s from seafood or fish oil help do the same thing, as we’ll explain.

3. Fishy omega-3s and antioxidant-rich foods spark fat-burning
Medical authorities use to assert that all calories were equal when it came to weight gain or loss, saying “a calorie is a calorie”.

That bit of conventional wisdom now lies in the dustbin of biomedical history, because it’s become clear that different foods exert very different effects on your metabolism.

Let’s take a look at two categories of foods that appear to spark fat burning: Antioxidants from plant foods, and omega-3 fats from seafood.

Foodborne antioxidants
Growing evidence suggests that certain dietary antioxidants spark fat-burning to a measurable extent.

This appears especially true of the flavonoid-type antioxidants that abound in colorful, non-starchy vegetables, chili peppers, tea (black or green), coffee, and chocolate (especially bars with 70% cocoa solids or more).

To learn more, see Can Peppers Aid Weight Control?, Does Colorful Produce Cut Pounds?, and
Food Allies in the Weight War: Spices, Tea, and Fish.

Seafood-source omega 3 fatty acids
Fat contains twice as many calories, compared with carbs or protein.

So adding fat to your diet can be counterproductive unless you eliminate a comparable quantity of calories derived from starchy, refined grain foods like bread, white rice, potatoes, pasta, and pastries.

But there’s good evidence that frequent enjoyment of fish — in place of starchy carbs — gives your metabolism a modest but significant boost.

Why would omega-3s boost your metabolism?

The answer stems from the fact that your body contains two basic types of fat:

  • White fat — This type accumulates in response to excess dietary calories and can emit pro-inflammatory chemicals. This is especially true of the larger white fat cells found in overweight people.
  • Brown fat — This type is less common, but its special cells host more of the fat-burning “factories” called mitochondria.

Once thought exclusive to babies, we now know that small amounts of brown fat occur in everyone — and people with a lower body mass index (BMI) tend to have more of it.

And growing evidence suggests that seafood-source omega-3s prompt the body to convert generally unhealthful white fat into fat-burning brown fat.

Back in 2015, we reported on Japanese research showing that in mice, supplemental omega-3 fish oil caused the animals’ “white” fat-storage cells to become “brown” or “beige” fat-burning cells … see Fish Oil Fuels Fat-Burning in Mice, and its links to our reports on supportive clinical and lab research.

Two recent studies from Spain and Texas confirm that — at least in mice — omega-3 fatty acids trigger the conversion of fat-storage cells into fat-burning cells (Quesada-López T et al. 2016; Pahlavani M et al. 2017).

Better yet, studies in mice suggests that omega-3s reduce the release of pro-inflammatory chemicals — which promote obesity, diabetes, and more — from white fat.

Other research shows that by supplementing with fish oil or eating omega-3 rich fish, participants add muscle mass (which boosts metabolism) and reduce body fat.

It’s believed that this effect may be due — at least in part — to fish oil’s ability to lower the levels of enzymes involved in storage of calories as fat.

Of course, we also need seafood-form omega-3s — DHA and EPA — for critical brain, eye, and immune functions.

(The plant-source omega-3 called ALA can’t serve these purposes until the body converts it — very inefficiently — into much smaller amounts of DHA and EPA.)

All wild seafood provides significant amounts of omega-3 DHA and EPA, but they’re especially abundant in fatty cold-water wild fish like salmon, tuna, sablefish, mackerel, and sardines (makes no major difference whether they're fresh, frozen, or canned).

You should avoid farm-raised fish, which are high in omega-6 fats, and (excepting farmed salmon) low in omega-3s.

See Tilapia Taken to Task, Farmed Fish Possess Unhealthful Fat Profiles, and Farmed Salmon's Diet Yields Unhealthful Cardiovascular Effects.

For more on this topic, see Omega-6 Fats Drive Obesity; Omega-3s Help Avoid ItOmega-3s Advised for Burning Fat and Curbing Appetite and Exercise + Omega-3s = Perfect Weight Loss Pair.

4. Favor protein over carbs
Among other effects, protein aids weight control by making you feel fuller faster and longer.

And diets rich in protein help preserve muscle, which helps retain your metabolic rate as you age.

(The purported benefits of higher-protein diets were confirmed recently in an unusually well-designed clinical trial: see Low-Carb Diet Won for Weight Loss and Health.)

We’re not necessarily talking about meat, poultry, and seafood, although they top the list of foods providing the most protein per pound.

Vegetables — especially colorful, non-starchy, choices like greens, broccoli, peppers, and onions — can provide significant amounts of protein, and most of their carbs are actively healthful ones.

The same is true of beans and nuts, which is why a vegetarian diet can provide sufficient protein, albeit much less reliably than omnivorous diets that include some meat, poultry, or seafood.

Your body uses about 10 percent of dietary calories for the digestive process. Because it takes more time and energy to digest protein, you get a little metabolic boost from high-protein meals.

The value of higher protein diets has been confirmed by clinical trials in overweight and obese men and women who’d been placed on low-calorie diets.

In the relevant trials, researchers placed participants on low-calorie diets, and assigned them either to high-protein menus or normal-protein menus — each menu providing the same number of total calories — with the high-protein groups eating fewer calories from carbs.

Encouragingly, the participants assigned to high-protein diets reported feeling fuller after meals, for longer periods, and a greater sense of satisfaction.

Better yet, the people assigned to the high-protein diets preserved more muscle, which was being consumed by all of the participants’ bodies, to compensate for getting too few calories to maintain their basal metabolic rate (BMR).

5. Fuel up every morning
Your mother was right – breakfast may just be the most important meal of the day!

Starting your day with a nutritious meal helps to stimulate digestion and get your metabolism going.

The National Weight Control Registry tracks thousands of people. And among those who’d lost an average of 60-plus pounds and kept it off for more than five years, nearly 80% ate a morning meal every day.

So eating breakfast seems to stimulate your metabolism — particularly if it includes a cup of coffee or tea.

And eating higher-protein/lower-carb breakfasts should help maintain muscle mass, boost your metabolism, and discourage storage of calories as fat.

We recommend morning meals that feature DHA-enriched eggs, bacon (preferably lower-sodium, from pasture-raised pigs), salmon sausage, salmon bacon, or smoked fish.

By all means, enjoy breakfast featuring whole grain cereals with fruits, nuts, and full-fat yogurt, but to maximize metabolic rate, don’t make such meals your exclusive morning routine.

For more on the benefits of breakfast — especially ones that feature protein and fat versus carbs — see High-Protein Breakfast Helps Blood Sugar Control, Can Paleo-Style Breakfasts Curb Food Cravings?, Protein for Breakfast Allays Appetites All Day, and Fat for Breakfast May Deter Diabetes.

 Note: This article originally appeared in March, 2017.

Sources


• Ahima RS. Adipose tissue as an endocrine organ. Obesity (Silver Spring). 2006 Aug;14 Suppl 5:242S-249S. Review.
• Buckley JD, Howe PR. Anti-obesity effects of long-chain omega-3 polyunsaturated fatty acids. Obes Rev. 2009 Nov;10(6):648-59. doi: 10.1111/j.1467-789X.2009.00584.x. Epub 2009 May 12. Review.
• Byrne HK, Wilmore JH. The effects of a 20-week exercise training program on resting metabolic rate in previously sedentary, moderately obese women. Int J Sport Nutr Exerc Metab. 2001 Mar;11(1):15-31.
• Clifton PM, Bastiaans K, Keogh JB. High protein diets decrease total and abdominal fat and improve CVD risk profile in overweight and obese men and women with elevated triacylglycerol. Nutr Metab Cardiovasc Dis. 2009 Oct;19(8):548-54. doi: 10.1016/j.numecd.2008.10.006.
• Flachs P, Rossmeisl M, Bryhn M, Kopecky J. Cellular and molecular effects of n-3 polyunsaturated fatty acids on adipose tissue biology and metabolism. Clin Sci (Lond). 2009 Jan;116(1):1-16. doi: 10.1042/CS20070456. Review.
• Galic S, Oakhill JS, Steinberg GR. Adipose tissue as an endocrine organ. Mol Cell Endocrinol. 2010 Mar 25;316(2):129-39. doi: 10.1016/j.mce.2009.08.018. Review.
• Garaulet M, Hernandez-Morante JJ, Lujan J, Tebar FJ, Zamora S. Relationship between fat cell size and number and fatty acid composition in adipose tissue from different fat depots in overweight/obese humans. Int J Obes (Lond). 2006 Jun;30(6):899-905.
• Geliebter A, Maher MM, Gerace L, Gutin B, Heymsfield SB, Hashim SA. Effects of strength or aerobic training on body composition, resting metabolic rate, and peak oxygen consumption in obese dieting subjects. Am J Clin Nutr. 1997 Sep;66(3):557-63.
• Howe P, Buckley J. Metabolic health benefits of long-chain omega-3 polyunsaturated fatty acids. Mil Med. 2014 Nov;179(11 Suppl):138-43. doi: 10.7205/MILMED-D-14-00154.
• Hunter GR, Byrne NM, Gower BA, Sirikul B, Hills AP. Increased resting energy expenditure after 40 minutes of aerobic but not resistance exercise. Obesity (Silver Spring). 2006 Nov;14(11):2018-25.
• Kamada, I, Truman L, Bold J, Mortimore D. (2011). The impact of breakfast in metabolic and digestive health. Gastroenterology and Hepatology From Bed to Bench, 4(2), 76–85.
• Kim M, Goto T, Yu R, Uchida K, Tominaga M, Kano Y, Takahashi N, Kawada T. Fish oil intake induces UCP1 upregulation in brown and white adipose tissue via the sympathetic nervous system. Sci Rep. 2015 Dec 17;5:18013. doi: 10.1038/srep18013.
• Labayen I, Díez N, González A, Parra D, Martínez JA. Effects of protein vs. carbohydrate-rich diets on fuel utilisation in obese women during weight loss. Forum Nutr. 2003;56:168-70.
• Laskowski ER/Mayo Clinic. Is body weight training effective as a strength training exercise? Accessed at http://www.mayoclinic.org/healthy-lifestyle/fitness/expert-answers/body-weight-training/faq-20147966
• Leidy H, Carnell N, Mattes R, Campbell W. Higher protein intake preserves lean mass and satiety with weight loss in pre-obese and obese women. Obes Res. 2007;15:421-429.
• Leidy HJ, Carnell NS, Mattes RD, Campbell WW. Higher protein intake preserves lean mass and satiety with weight loss in pre-obese and obese women. Obesity (Silver Spring). 2007 Feb;15(2):421-9.
• Leidy HJ, Mattes RD, Campbell WW. Effects of acute and chronic protein intake on metabolism, appetite, and ghrelin during weight loss. Obesity (Silver Spring). 2007 May;15(5):1215-25.
• Leidy HJ, Tang M, Armstrong CL, Martin CB, Campbell WW. The effects of consuming frequent, higher protein meals on appetite and satiety during weight loss in overweight/obese men. Obesity (Silver Spring). 2011 Apr;19(4):818-24. doi: 10.1038/oby.2010.203.
• McDonald C, Bauer J, Capra S, Waterhouse M. Muscle function and omega-3 fatty acids in the prediction of lean body mass after breast cancer treatment. Springerplus. 2013 Dec 19;2:681. doi: 10.1186/2193-1801-2-681. eCollection 2013.
• McDonald C, Bauer J, Capra S. Omega-3 fatty acids and changes in LBM: alone or in synergy for better muscle health? Can J Physiol Pharmacol. 2013 Jun;91(6):459-68. doi: 10.1139/cjpp-2012-0304. Epub 2012 Nov 21.
• Moreno-Aliaga MJ, Lorente-Cebrián S, Martínez JA. Regulation of adipokine secretion by n-3 fatty acids. Proc Nutr Soc. 2010 Aug;69(3):324-32. doi: 10.1017/S0029665110001801. Review.
• Pahlavani M, Razafimanjato F, Ramalingam L, Kalupahana NS, Moussa H, Scoggin S, Moustaid-Moussa N. Eicosapentaenoic acid regulates brown adipose tissue metabolism in high-fat-fed mice and in clonal brown adipocytes. J Nutr Biochem. 2017 Jan;39:101-109. doi: 10.1016/j.jnutbio.2016.08.012.
• Parker B, Noakes M, Luscombe N, Clifton P. Effect of a high-protein, high-monounsaturated fat weight loss diet on glycemic control and lipid levels in type 2 diabetes. Diabetes Care. 2002 Mar;25(3):425-30.
• Quesada-López T, Cereijo R, Turatsinze JV, Planavila A, Cairó M, Gavaldà-Navarro A, Peyrou M, Moure R, Iglesias R, Giralt M, Eizirik DL, Villarroya F. The lipid sensor GPR120 promotes brown fat activation and FGF21 release from adipocytes. Nat Commun. 2016 Nov 17;7:13479. doi: 10.1038/ncomms13479.
• Raclot T, Groscolas R, Langin D, Ferré P. Site-specific regulation of gene expression by n-3 polyunsaturated fatty acids in rat white adipose tissues. J Lipid Res. 1997 Oct;38(10):1963-72
• Sakurai T, Ogasawara J, Shirato K, Izawa T, Oh-Ishi S, Ishibashi Y, Radák Z, Ohno H, Kizaki T. Exercise Training Attenuates the Dysregulated Expression of Adipokines and Oxidative Stress in White Adipose Tissue. Oxid Med Cell Longev. 2017;2017:9410954. doi: 10.1155/2017/9410954. Review.
• Schwecherl L. 50 Bodyweight Exercises You Can Do Anywhere. December 17, 2014. Accessed at http://greatist.com/fitness/50-bodyweight-exercises-you-can-do-anywhere
• Sedano S. The 20 Best Body-Weight Exercises. Feb 17, 2017. Accessed at http://www.livestrong.com/slideshow/551492-2o-best-body-weight-exercises/
• Takahashi Y, Ide T. Dietary n-3 fatty acids affect mRNA level of brown adipose tissue uncoupling protein 1, and white adipose tissue leptin and glucose transporter 4 in the rat. Br J Nutr. 2000 Aug;84(2):175-84.
• Tang M, Armstrong CL, Leidy HJ, Campbell WW. Normal vs. high-protein weight loss diets in men: effects on body composition and indices of metabolic syndrome. Obesity (Silver Spring). 2013 Mar;21(3):E204-10. doi: 10.1002/oby.20078.
• Todoric J, Löffler M, Huber J, Bilban M, Reimers M, Kadl A, Zeyda M, Waldhäusl W, Stulnig TM. Adipose tissue inflammation induced by high-fat diet in obese diabetic mice is prevented by n-3 polyunsaturated fatty acids. Diabetologia. 2006 Sep;49(9):2109-19.